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By: Joseph A. Smith, Jr., MD

• Professor of Urologic Surgery, Vanderbilt University, Nashville, Tennessee

A few instances might be discovered to medicine 5325 100 mg epitol sale be because of medicine 44 159 purchase 100 mg epitol with visa a toxic myopathy medicine allergy purchase 100mg epitol with amex, significantly from one of the statin class of medication medications pancreatitis cheap epitol 100mg otc. Hypothyroidism, McArdle illness, hyperparathyroidism, steroid myopathy, adrenal insufficiency, hyperinsulinemia, and early rheumatoid arthritis must be dominated out by acceptable studies (these toxic and metabolic myopathies are mentioned in Chap. Once recovery has begun, usually several weeks or longer, and the dosage could also be reduced steadily toward 20 mg daily. It is then acceptable to attempt to control the illness with an alternate-day schedule with double this quantity. After cautious discount of prednisone over a interval of 6 months to a year or longer, the patient can often be maintained on doses of seven. In acute and significantly severe instances, treatment could also be facilitated by the use initially of excessive-dose "pulse" methylprednisolone (l g infused over 2 h each day for 3 to 5 days). This form of treatment should be regarded as a temporary measure until oral prednisone becomes effective. Alternatively, or generally in tandem with this approach, intravenous immunoglobulin or plasma exchanges could also be instituted. Currently favored as an adjunct to steroids is methotrexate (5 to 10 mg/week in three divided oral doses, increased by 2. Methotrexate or azathioprine ought to generally be given along with the smallest effective doses (15 to 25 mg) of prednisone. The steroid-sparing results of azathioprine may also be helpful in sufferers requiring lengthy-time period remedy. Cyclosporine has additionally been utilized in recalcitrant instances; it has few advantages over different immunosuppressant medication and has a number of doubtlessly critical unwanted effects, together with nephrotoxicity. Whole-physique radiation is said to be useful in a few drug-resistant instances, but the unwanted effects should be thought-about. Several of our sufferers have had severe aspiration pneumonias because of their dysphagia. The interval of activity of illness varies significantly but is often around 2 years in both the kid and grownup. Approximately 20 percent of our sufferers have recovered completely after association of steroid remedy, and longterm remissions have been achieved in about an equal quantity. The extent of recovery is roughly proportional to the acuteness and severity of the illness and the duration of symptoms prior to establishment of remedy. In the series of DeVere and Bradley, by which sufferers were treated early, there was remission in over 50 percent of instances, whereas within the series of Riddoch and Morgan-Hughes, by which sufferers were treated more than 2 years after onset of the illness, the rate was significantly decrease. Even in sufferers with a coexistent malignancy, muscle weak point might lessen and serum enzyme ranges decline in response to corticosteroid remedy, but weak point returns after a few months and will then be proof against additional treatment. As already acknowledged, if the tumor is successfully removed, the muscle symptoms might remit but even this expertise has not been uniform. Clinical Manifestations the sickness is characterized by a steadily progressive, painless muscular weak point and atrophy, which can be generalized or affect the limbs selectively and often asymmetrically. Either the proximal or distal muscle tissue could also be affected, or the limbs could also be involved kind of evenly. In about 20 percent of instances the illness begins with focal weak point of the quadriceps, finger or wrist flexors, or decrease leg muscle tissue on one or each side and spreads to different muscle groups solely after many months or years. Selective weak point of the flexor pollicis longus is a particularly characteristic sample of involvement, and isolated quadriceps weak point or neck extensor weak point are different patterns that should bring the diagnosis to thoughts. The tendon reflexes are normal initially but diminish in about half the sufferers as the illness progresses. The selective or uneven involvement of distal muscle tissue might suggest the faulty diagnosis of motor neuron illness. The diagnosis in the end is dependent upon nearly diagnostic features within the muscle biopsy. Also, the medical context by which these different diseases happen often trigger little problem in figuring out the inclusions as an ancillary and minor abnormality. Its defining features, the intracytoplasmic and intranuclear inclusions, were first described in 1965 by Adams and colleagues, who additionally drew consideration to a number of medical attributes now thought-about characteristic.

Agraphia medications quiz discount epitol 100mg with amex, Alexia xanthine medications cheap 100 mg epitol with visa, Acalculia medicine 1920s effective epitol 100mg, Apraxia Anterior alexia Sites of lesions inflicting agraphia Central alexia Topography of lesions in alexia Numerical alexia/agraphia medications to treat bipolar disorder order 100 mg epitol otc, anarithmia Sites of lesions inflicting acalculia Ideomotor apraxia Dressing apraxia Lid-opening apraxia 129 Rohkamm, Color Atlas of Neurology © 2004 Thieme All rights reserved. Behavioral Manifestations of Neurological Disease Speech Disorders Creation of the sounds of speech (articulation). The sounds of speech are created by altering the configuration of the physiological resonance areas and articulation zones. The resonance areas could be altered by motion of the velum (which separates the oral and pharyngeal cavities) and tongue (which divides the oral cavity). Each vowel (a, e, i, o, u) is associated with a specific partitioning of the oral cavity by the tongue. The palate, tooth, and lips are the articulation zones with which consonants are produced (g, s, b, and so forth. Behavioral Manifestations of Neurological Disease Speech the neural basis of speech. Speech-associated motion applications generated in the premotor cortex (space 6) are modulated by info from the cerebellum and basal ganglia and are relayed to the motor cortex (inferior portion of the precentral gyrus, space 4) for implementation. The motor cortex initiatives by the use of the corticopontine and corticobulbar tracts to the motor cranial nerve nuclei in the brain stem. Speech-associated impulses to the respiratory muscles travel (amongst different pathways) from the motor cortex to the spinal anterior horn cells. Connections to the basal ganglia and cerebellum are necessary for the coordination of speech. These impulses are processed by a neural network (reticular formation, thalamus, precentral cortex) mediating suggestions control of speech. The central innervation of the speech pathway is predominantly bilateral; thus, dysarthria as a result of unilateral lesions is normally transient. Voice production by the larynx (phonation) by way of the vibrating vocal folds (cords) yields sound at a basic frequency with a various admixture of higher-frequency components, which lend the voice its timbre (musical quality); timbre depends on the resonant cavities above the vocal folds (pharynx, oral cavity, nasal cavity). The volume of the voice is regulated by stretching and rest of the vocal folds and by adjustment of air pressure in the larynx. The air move necessary for phonation is produced in the respiratory tract (diaphragm, lungs, chest, and trachea). Dysarthria, Dysphonia Dysarthria (impaired articulation) and dysphonia (impaired phonation and resonance) end result from a disturbance of the neural control mechanism for speech (sensory portion, motor portion, or each). Speech Disorders Thalamocortical projections Motor cortex Cerebellum Corticobulbar fibers Basal ganglia Trigeminal n. Behavioral Manifestations of Neurological Disease Disturbances of Orientation Agnosia is outlined as a disturbance of recognition by which notion, consideration, and general intelligence are (largely) unimpaired. Constructional apraxia is characterized by the lack to represent spatial relationships in drawings, or with building blocks. Everyday activities are impaired by the lack to draw diagrams, read (analog) clocks, assemble items of apparatus or tools, or write phrases in the correct order (spatial agraphia). Hemineglect is the lack to consciously perceive, react to, or classify stimuli on one side in the absence of a sensorimotor deficit or exceeding what one would count on from the severity of the sensorimotor deficit present. Hemineglect could involve unawareness of one side of the physique (one-sided tooth brushing, shaving, and so forth. Neurological examination reveals that double simultaneous stimulation (touch, finger motion) of homologous physique parts (same site. The accountable lesion is normally, although not all the time, in the temporoparietal area (angular and supramarginal gyri). Anosognosia is the unawareness or denial of a neurological deficit, corresponding to hemiplegia. Anosognosia may accompany visual subject defects as a result of unilateral or bilateral lesions of the visual cortex (homonymous hemianopsia, cortical blindness). The most putting example of that is Anton syndrome, by which cortically blind patients act as if they may see, and will even "describe" details of their surroundings (incorrectly) without hesitation. Disturbances of Orientation Hemispatial neglect (left side) (task was to draw a clockface and set it to "quarter past 12") Hemispatial neglect (left side) 133 Rohkamm, Color Atlas of Neurology © 2004 Thieme All rights reserved. Behavioral Manifestations of Neurological Disease Disturbances of Memory 5­10 minutes later.

See additionally particular medication movement problems due to medications a to z buy cheap epitol 100mg online medications neuropathy purchase 100 mg epitol with visa, 64 Phenothiazine derivatives symptoms 7 days after iui quality 100mg epitol, retinal degeneration associated with treatment laryngomalacia infant quality 100 mg epitol, 211 Phenylalanine hydroxylase deficiency. See additionally particular problems back ache associated with, 182­ 183 fatigue and lassitude as signs of, 434 with head injury, 764­ 765 headache with, 158 neurotic. See Neuroses; particular problems ache associated with, 122 patient method and, eight psychotic. See Psychoses; particular problems with seizures, 278­ 279 sleep disturbance due to, 339­ 340 Psychiatric dizziness, 258 Psychogenic seizures, 273­ 274 Psychologic disturbance, sleep disturbance due to, 339 Psychometry, 33 Psychomotor exercise, reduced, acute confusional states associated with, 360­ 361, 361t Psychomotor asthenia, 552 Psychomotor retardation, 359 Psychomotor seizures, 277­ 279, 279f Psychomotor triad, 278 Psychopathy, 1291 Psychosensorimotor regression, in infants, 805 Psychoses, 1308­ 1316. See ArgyllRobertson pupils in comatose patient, 313 Marcus-Gunn pupillary signal and, 242, 1184 in microphthalmia with corneal opacities, eccentric pupils, spasticity, and severe mental retardation, 857 pupillary light reflex and, 241, 241f springing, 243 Pupillary defect, afferent, 206 Pupillary denervation, analysis of, 460t, 461 Pupillary light reflex, 241, 241f Pupilloconstrictor fibers, 241 Pupillodilator fibers, 241 Pure motor hemiplegia, 682 Pure pandysautonomia, 462­ 463, 1130 Pure sensory stroke, 682 Pure word-blindness, 418t, 422 Pure word-deafness, 253, 409­ 410, 418t, 421 Pure word mutism, 418t, 419, 422 Purkinje cells, 72, seventy four Purpura mind, 727 thrombotic thrombocytopenic, 736 Pursuit actions, 222­ 223 Putaminal hemorrhage, 713 Pyle syndrome, 1159 Pyramidal tract, 41, 42f, 43f. See additionally particular patterns afferent influences on, 473­ 474 apneustic, in comatose sufferers, 315 Biot, in comatose sufferers, 315 blood strain and, 459, 460t central motor mechanisms of, 472f, 472­ 475 in comatose sufferers, 315 ventilatory problems due to neurologic disease and, 475 voluntary control of, 473 Respiratory alternans, 475 Respiratory muscular tissues, weak point of, 1196 Rest tremor, eighty three pathophysiology of, eighty five Restless legs syndrome, ninety seven, 339 Reticulospinal tract, forty five Retina, 205, 206f abnormalities of, 207­ 211 cherry-red spot in, in Tay-Sachs disease, 807, 807f degenerations of, 211 hemorrhages of, 208­ 209 Retinal artery, central, 207 occlusion of, 209, 209f, 210f Retinal degeneration, paraneoplastic, 583t Retinal detachment, 205 Retinal diseases, hereditary listening to loss with, 951 Retinal migraine, 151 Retinal vein, thrombosis of, 210, 210f Retinal vessels. See additionally Retinal artery; Retinal vein changes in, 208 Retinitis pigmentosa, 211, 950 Retinoblastomas, 567 Retinopathy degenerative, 211 diabetic, 211 hypertensive, 728 paraneoplastic, 587 serous, 210 Retrobulbar neuritis, 213­ 215, 214f, 214t in a number of sclerosis, 777­ 778 Retrocollis, 91 Retrograde amnesia, 357, 376 with concussion, 751 in Wernicke-Korsakoff syndrome, 985 Retrolental fibrodysplasia, 857 Retroviral an infection. See additionally particular medication and drug varieties Seesaw nystagmus, 239 Segawa disease, sixty seven, 930­ 931 Segmental demyelination, 1111­ 1112, 1112f Segmental myoclonus, 89 Seizures. See additionally Aging; Elderly folks Senile chorea, 64 Senile gait, 102t, 106­ 107, 107f Senile plaques, 901, 902 Senile tremor, 81t, 81­ eighty two, 82f pathophysiology of, eighty five treatment of, eighty two­ eighty three Sensation, 129f, 129­ 142. See Sensory exams sensory pathways and, 131­ 133, 132f, 134f specificity theory of, one hundred thirty terminology and, 135 Sensitization, 117 Sensorimotor neuropathic paralysis, subacute, 1130­ 1137 in diabetic neuropathy, 1134­ 1137 in nutritional deficiency neuropathy, 1130­ 1131 in paraneoplastic polyneuropathy and sensory ganglionopathy, 1131­ 1132 in subacute toxic neuropathies, 1132­ 1134 Sensorimotor polyneuropathy, a number of symmetrical lipomas with, 1151 Sensorimotor spinal tract syndrome, 1081 Sensorineural deafness, 248, 253­ 256 hereditary, 253, 254t­ 255t, 256 Sensory operate improvement of, 500­ 501 delays in, 506 in peripheral nerve disease, 1114 Sensory ganglionopathy, 1128, 1131­ 1132 idiopathic, 1141­ 1142 Sensory loss, hysterical, 1299 Sensory nerve(s), 131 Sensory nerve motion potentials, 1099 Sensory neuritis, migrant, 1142 Sensory neuronopathy, 1116­ 1117 paraneoplastic, 583t, 586 Sensory neuropathy, acute, 1128 Sensory paroxysms, 342 Sensory perineuritis, 1142 Sensory polyneuropathy continual, 1143­ 1151 acquired forms of, 1144­ 1151 genetic forms of, 1151­ 1163, 1152t­ 1154t continual delicate, of elderly, 1151, 1151t hereditary, mutilating, in adults, 1156 mutilating, recessive, of childhood, 1156 with paraproteinemia, 1144 Sensory seizures, 276­ 277 Sensory syndromes, 138­ 142 with brainstem lesions, 141 cortical, four hundred­ 401 analysis of, 142 due to suggestibility and hysteria, 142 involving nerve roots, 139. See additionally Radiculopathy involving peripheral nerves, 138­ 139 a number of, 138­ 139, 139t. See additionally Ganglionopathy; Neuronopathy with parietal lesions, 142 spinal, 140f, one hundred forty­ 141 with thalamic lesions, 141­ 142 Sensory exams, 7, 133­ 138, 134t of deep-strain ache, 136 of discriminative sensation, 137­ 138 of ache perception, 135­ 136 of proprioceptive sense, 136 of tactile sensation, 135 of thermal sense, 136 of vibratory sense, 136­ 137 Septic encephalopathy, 363, 971 Septic thrombophlebitis, intracranial, 605­ 606 Septo-optic dysplasia, 857 Serologic exams, of cerebrospinal fluid, 15 Serotonergic fibers, 75 Serotonergic neurons, sleep and, 336 Setting-sun signal, 225, 534 Seventh nerve. See additionally Drowsiness daytime, excessive, 344­ 345 Sleeping illness, 340, 344, 625 Sleep-wake pattern disturbances of, 341 reversal of, 344 Slit ventricle syndrome, 537 Slow channel syndrome, 1260t, 1262 Slurring dysarthria, seventy seven Smell sense, 195­ 199 anatomy and physiology of, 195­ 197, 196f disturbances of, 197t, 197­ 199, 398 Smith-Lemli-Opitz syndrome, 857­ 858 Smith-Magenis syndrome, 858 Snake venom toxins, 1260t Sneddon syndrome, stroke complicating, 736 Snellen chart, 203­ 204 Social habits improvement of, 504 disturbances of, in acute confusional states, 359 Sociopathy, 446, 513, 1291, 1302­ 1304 Sodium benzoate, for hyperglycinemia, 802 Sodium channel diseases, 286t, 1266t, 1268­ 1270 Sodium nitroprusside, for hypertensive encephalopathy, 729 Sodium valproate, for palatal tremor, 86 for seizures, 294­ 295 for trigeminal neuralgia, 163 Solutes, in cerebrospinal fluid, 15­ 16 Somatosensory cortex, 399 Somatosensory evoked potentials, 1100­ 1101 Somatosensory seizures, 276 Somnambulism, 343 Somnolence, with bulimia, 488 Somnolescent starts, 341­ 342 Spasm(s) back ache with, one hundred seventy convergence, 1299 habit in akathisia, ninety seven in Tourette syndrome, 95­ ninety seven hemifacial, 1184 intention, sixty five lingual, 94 masticatory, 94 occupational, 94 oromandibular, 94 in peripheral nerve disease, 1116 phonatory, 931 respiratory, 931 in tetanus, 1030­ 1031 torsion. See Dystonia Spasmodic dysphonia, 67t, 91, 428­ 429 Spasmodic laughing/crying, 445t, 445­ 446, 450 Spasmodic torticollis, 91, 92f, 93 Spasmus mutans, 238 Spastic and ataxic syndrome, spinal, nutritional, 991 Spastic diplegia, 876 Spastic dysarthria, 426­ 427 Spastic dysphonia, 931 Spastic gait, 102t, 104 Spastic paraparesis, tropical, 1059 Spastic paraplegia familial, 1084 hereditary forms of, 947­ 948, 948t polyneuropathy with, 1157 Spasticity, forty six clasp-knife, 63 Specificity theory of ache, 111 of sensation, one hundred thirty Speech, 413. See additionally Language features; Speech and language problems Speech and language problems, 417­ 428 agraphias as, 423­ 424 aphasia as, 417­ 421, 418t, 424 method to patient with, 424­ 426 of articulation and phonation, 426­ 428 developmental, 506­ 511 in disconnection syndromes, 421­ 423 Speech discrimination test, 250­ 251 Spelling difficulty, 511 Sphenoid ridge, meningioma of, 577 Sphingolipidoses, in infants, 805, 806t Sphingomyelinase deficiency. See Werdnig-Hoffmann disease Spinal myoclonus, 89 Spinal neuronitis, subacute, 89, 1066­ 1067 Spinal osteomyelitis, tuberculous, 1061, 1062f Spinal puncture headache, 159 Spinal roots, 131, 132f Spinal shock, forty six, 465, 1051­ 1052 Spinal spastic and ataxic syndrome, nutritional, 991 Spinal standing, 1052 Spinal subdural hemorrhage, 1072 Spinal tract syndrome, sensorimotor, 1081 Spinal tumors, 1079­ 1083 anatomy and, 1079­1081, 1080f­1082f differential analysis of, 1082­ 1083 intramedullary metastases, 1081, 1082f primary, 1080f, 1080­ 1081 secondary, 1081, 1081f symptomatology of, 1081­ 1082 treatment of, 1083 Spindle coma, 307 Spine. See additionally particular areas of spine angiography of, 21­ 22 computed tomography of, 18, 18f magnetic resonance imaging of, 18­ 21, 19t, 20f­ 22f myelography of, contrast, 18 Paget disease of, 1077 rheumatoid arthritis of, 184 Spinocerebellar degeneration, 831­ 834 Spinocerebellar tract dorsal, 71 ventral, 71 Spinocerebellum, 71 Spinoreticulothalamic tract, one hundred fifteen, 116 Spinothalamic tract, lateral, 113, 115f, 116f Spinothalamic tractotomy, for ache, 126 Spirometria, 626t Spironolactone, for hypokalemic periodic paralysis, 1271 Spondylolisthesis, 172, 173 intact arch, 173 lumbar stenosis and, a hundred and eighty Spondylolysis, 173 Spondyloptosis, 173 Spondylosis cervical. See Cervical spondylosis lumbar, 1076 Spongiform encephalopathies, transmissible, 653­ 656. See additionally Corticosteroid entries Steroid myopathy, continual, 1237 Stevens-Johnson syndrome, 204, 733 Stickler syndrome, 254t Stiff-infant syndrome, 1279 Stiff-man syndrome, 238, 1067, 1105, 1199, 1279 paraneoplastic, 583t, 587 Stilbamidine, peripheral neuropathy due to, 1133 Stimulant medication, 1028 Stings, 1033­ 1034 Stokes-Adams faints, 327 Stomach carcinoma of, back ache with, 182 gastric hemorrhage and, 488 Stomatodynia, a hundred sixty five Strabismus, 229­ 230 alternating, 230 comitant, 230 concomitant, 222 nonparalytic, 229 paralytic, 229 Strachan syndrome, 216, 992 Straight-leg raising test, 171 Strain, of lower back, 173­ 174 Stress, 438, 439 autonomic operate exams and, 450­461, 460t Striate cortex, 404 Striatocapsular infarction, 672 Striatonigral degeneration, 463, 925 Stroke. See additionally Neurovascular syndromes with migraine, 151­ 152 risk elements for, 663­ 664 seizures following, 740 sensory, pure, 682 temporal profile of, 662 Strumpell-Lorrain disease, 947­ 948, Ё 948t Stump neuromas, 189 Stupor, 304 acquired metabolic diseases presenting as syndrome of, 959­ 975 alcoholic, 1006­ 1007 hepatic, 29, 967­ 969 recurring, 309 Sturge-Weber syndrome, 872 Stuttering, 508­ 509 acquired, 427 St. See Hydrocephalus, obstructive (tension) Teratogens, 865 Teratomas, 569, 570 Tetanus, 1030­ 1031, 1280 cephalic, 1031 Tetany, 1237­ 1238, 1277 Tethered cord, 860­ 861, 861f myelopathy and, 1078 Tetrabenazine, for palatal tremor, 86 Tetracycline for Lyme disease, 620 for neurosyphilis, 618 Tetraplegia. See Dystonia Torticollis, 931 congenital, 1245 spasmodic, 91, 92f, 93 Tortopia, 236 Torulosis, 621­ 622 Tourette syndrome, 95­ ninety seven, 1296 Tower skull, 853 Toxic encephalopathies, aggressive habits in, 448 Toxic neuropathies optic, 215­ 216, 216t subacute, 1132­ 1134 Toxic oil poisoning, 1038 Toxic polyneuropathy, acute, 1129­ 1130 Toxic vestibulopathy, 263 Toxins. See Brain tumors; Metastatic disease; Neoplasms; Paraneoplastic problems; Spinal tumors; particular tumors Turner syndrome, 864 Turricephaly, 853 Tussive syncope, 326 Twilight states, 356 Twitch-convulsive syndrome, uremic, 969­ 971 Two-point discrimination, testing of, 137 Typhus epidemic, 623 inflammatory vasculitis in, 730 murine (endemic), 623 scrub, 623 Tyrosine hydroxylase deficiency, 818 Tyrosinemia, hereditary (oculocutaneous), 817­ 818 U Uhthoff phenomenon, 776 Ulnar nerve, mononeuropathy of, 1168 Ultrasound, of skull and spine, 23 Umami, 199 Uncal syndrome, 310 Uncinate seizures, 277 Unconsciousness. See additionally Coma in cerebrovascular disease, 738 definition of, 302­ 303 transient, following head injury, treatment of patient with, 765 Unilateral ataxia, seventy eight­ seventy nine Universal analgesia, 123 University of Pennsylvania Smell Identification Test, 197 Unverricht-Lundborg disease, 285 in late childhood and adolescence, 826 Upbeat nystagmus, 237­ 238 Upper motor neuron. See additionally particular infections fatigue following, 435­ 436 mechanisms of, 631­ 632 of muscle, 1203 myelitis due to, 1058­ 1059 pathways of an infection and, 631 retroviral, 644­ 648. See Ё Hepatolenticular degeneration Whiplash accidents, headache with, 158 Whipple disease encephalitis in, 603 vertical gaze palsy in, 227 Whiskey matches, 290, 1008­ 1009 Williams syndrome, 511, 864, 888 Wilson disease. See Hepatolenticular degeneration Wing-beating tremor, seventy six Withdrawal seizures, 290, 1008­ 1009 Withdrawal syndromes alcohol. See Cerebrohepatorenal disease Ziprasidone (Geodon), for schizophrenia, 1328t Zolmitriptan, for headache, 159 Zolpidem (Ambien), 1023 for insomnia, 340 Zona, 641 Zoster. C3 C3 C4 T2 C5 T3 T4 T5 T6 T7 T8 T9 T10 T11 T12 L1 C7 C8 S5 S4 L2 S3 C5 C6 T1 T2 T3 T4 T5 T6 T7 T8 T9 T10 T11 T12 L1 L L3 2 S1 S2 C4 C5 T2 T1 C6 C6 C8 S2 L3 L3 L4 L5 L4 L5 S1 S1 L5 Figure 9-2. The book demonstrates how treatment methods relate to the various theories as to why stuttering and cluttering come up, and how they develop.

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References:

• http://www.pkids.org/pdf/idw/text1.pdf
• https://www.gbiosciences.com/image/pdfs/protocol/JC-1_Mitochondrial_Membrane_Potential_Assay.pdf
• http://irjns.org/article-1-84-en.pdf
• https://downloads.hindawi.com/journals/crin/2014/792954.pdf
• https://www.northallegheny.org/cms/lib4/PA01001119/Centricity/Domain/1110/karyotyping%20lab%20KEY.pdf